Publication date: Jul 12, 2024
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) interacts between the host and virus and govern induction, resulting in multiorgan impacts. Its pathophysiology involves the followings: 1) the angiotensin-converting enzyme (ACE2) and Toll-like receptor (TLR) pathways: 2) the neuropilin (NRP) pathway: 3) the spike protein pathway. Therefore, it is necessary to block the pathological course with modulating innate lymphoid cells against diverse corona variants in the future.
| Concepts | Keywords |
|---|---|
| Ace2 | ACE2 |
| Biomed | COVID-19 |
| Coronavirus | SARS-CoV-2 |
| Host | Spike protein |
| Pathological | TLR |
Semantics
| Type | Source | Name |
|---|---|---|
| disease | VO | Severe acute respiratory syndrome coronavirus 2 |
| disease | IDO | host |
| drug | DRUGBANK | Angiotensin II |
| disease | VO | Toll-like receptor |
| disease | MESH | COVID-19 |
Original Article
(Visited 1 times, 1 visits today)