Systems vaccinology identifies immunological correlates of SARS-CoV-2 vaccine response in solid organ transplant recipients.

Publication date: Jul 01, 2025

Solid-organ transplant (SOT) recipients are at enhanced risk of infection and to poorly respond to vaccination due to comorbidities and immunosuppression. We performed a systems vaccinology study in 59 kidney and 31 lung transplant recipients who received 3 doses of COVID-19 mRNA BNT162b2 vaccine. We were able to characterize a baseline configuration associated with an effective humoral response to 3 doses, characterized by an innate and activated B cell profile, whereas a T cell signature was associated with a poorer response. We observed a distinct configuration associated with a detectable humoral response to 2 doses, partly mediated by double negative B cell subsets. These results suggest that, despite their immunosuppression, some SOT recipients can induce an effective humoral response to 3 doses of vaccine supported by a baseline configuration close to the healthy phenotype. Baseline immune phenotyping may help identify SOT recipients at the greatest risk of a poor vaccine response.

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Concepts Keywords
Bnt162b2 Baseline
Immunosuppression Configuration
Kidney Doses
Organ Effective
Poor Humoral
Immunosuppression
Organ
Recipients
Risk
Solid
Sot
Systems
Transplant
Vaccine
Vaccinology

Semantics

Type Source Name
disease MESH infection
disease IDO immunosuppression
disease MESH COVID-19
disease IDO cell
disease MESH cardiovascular disease
disease IDO susceptibility
pathway REACTOME Immune System
disease MESH Infectious Diseases
pathway REACTOME Adaptive Immune System
drug DRUGBANK Coenzyme M
disease MESH influenza
drug DRUGBANK Yellow Fever Vaccine
pathway REACTOME Innate Immune System
drug DRUGBANK Mycophenolate mofetil
drug DRUGBANK Monomethyl fumarate
drug DRUGBANK Esomeprazole
drug DRUGBANK Azathioprine
drug DRUGBANK Tacrolimus
drug DRUGBANK Ciclosporin
drug DRUGBANK Everolimus
drug DRUGBANK Serine
disease IDO blood
disease MESH Overweight

Original Article

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