Characterization of the SARS-CoV-2-Specific T Cell Responses in Rheumatoid Arthritis Subjects Vaccinated for COVID-19 Protection.

Characterization of the SARS-CoV-2-Specific T Cell Responses in Rheumatoid Arthritis Subjects Vaccinated for COVID-19 Protection.

Publication date: Dec 01, 2025

The efficacy of mRNA-based vaccines to prevent COVID-19 in autoimmune patients is controversial due to immunosuppressive therapies. Here, we characterized the T cell responses to SARS-CoV-2 in rheumatoid arthritis (RA) subjects, who received as few as one or up to seven vaccine injections. The study population had different disease severities and an association with other autoimmune comorbidities. All the subjects studied showed SARS-CoV-2 spike-specific CD4+ T helper (Th) cells in circulation, and in most of the subjects, CD8 cytotoxic T cells. CD4 and CD8 T cells were CCR6+, suggesting trafficking to tissues. T cell memory did not correlate with the number of vaccine boosts received. Nonspike-specific T cells were enumerated in subjects who had symptomatic or asymptomatic COVID-19. Spike- and nonspike-specific regulatory T cells (Treg) were detectable in most subjects. CD4 CD8 double-negative (DN) T cells expanded in response to SARS-CoV-2 peptides. DN T cells co-cultured with autologous myeloid dendritic cells (DC) differentiated into CD8 T cells, depending upon the strength of the stimuli. RA subjects responded to mRNA-based vaccination for COVID-19 protection, with no correlation with the number of injections. DN T cells differentiated into CD8 T cells after stimulation, potentially exacerbating inflammation in RA vaccine recipients.

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Concepts Keywords
Cd4 Adult
Cultured Aged
Immunosuppressive Arthritis, Rheumatoid
Mrna CD8-Positive T-Lymphocytes
COVID-19
COVID-19 Vaccines
COVID-19 Vaccines
Female
Humans
Immunologic Memory
Male
Middle Aged
regulatory T cells
SARS-CoV-2
Spike Glycoprotein, Coronavirus
Spike Glycoprotein, Coronavirus
spike protein, SARS-CoV-2
T cell memory
T-Lymphocytes, Regulatory
Vaccination

Semantics

Type Source Name
disease MESH Rheumatoid Arthritis
pathway KEGG Rheumatoid arthritis
disease MESH COVID-19
disease MESH inflammation
disease MESH Infection
disease MESH Infectious Diseases
drug DRUGBANK Tretamine
pathway REACTOME Reproduction
pathway REACTOME TCR signaling
disease MESH relapses
drug DRUGBANK Pentaerythritol tetranitrate
disease MESH Sjogren syndrome
disease MESH psoriasis
disease MESH Hashimoto thyroiditis
disease MESH asymptomatic infection
drug DRUGBANK Amino acids
disease MESH included
drug DRUGBANK Dimethyl sulfoxide
drug DRUGBANK Diatrizoate
disease MESH APC
disease MESH Star
disease MESH fibrosis
disease MESH syndrome
disease MESH systemic lupus erythematosus
pathway KEGG Systemic lupus erythematosus
disease MESH Tetanus
disease MESH Diphtheria
disease MESH Pertussis
pathway KEGG Pertussis
drug DRUGBANK Coenzyme M
disease MESH Arthritis
disease MESH Liver Diseases
disease MESH Lam
disease MESH Lung Inflammation
disease MESH Viral Infections
disease MESH Liver Fibrosis
disease MESH Alcoholic Steatohepatitis
drug DRUGBANK Chlorotrianisene
disease MESH Death
drug DRUGBANK Albendazole

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