Long COVID involves activation of proinflammatory and immune exhaustion pathways.

Long COVID involves activation of proinflammatory and immune exhaustion pathways.

Publication date: Dec 12, 2025

Long COVID (LC) involves a spectrum of chronic symptoms after acute severe acute respiratory syndrome coronavirus 2 infection. Current hypotheses for the pathogenesis of LC include persistent virus, tissue damage, autoimmunity, endocrine insufficiency, immune dysfunction and complement activation. We performed immunological, virological, transcriptomic and proteomic analyses from a cohort of 142 individuals between 2020 and 2021, including uninfected controls (n = 35), acutely infected individuals (n = 54), convalescent controls (n = 24) and patients with LC (n = 28). The LC group was characterized by persistent immune activation and proinflammatory responses for more than 180 days after initial infection compared with convalescent controls, including upregulation of JAK-STAT, interleukin-6, complement, metabolism and T cell exhaustion pathways. Similar findings were observed in a second cohort enrolled between 2023 and 2024, including convalescent controls (n = 20) and patients with LC (n = 18). These data suggest that LC is characterized by persistent activation of chronic inflammatory pathways, suggesting new therapeutic targets and potential biomarkers of disease.

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Concepts Keywords
Biomarkers Activation
Coronavirus Acute
Covid Chronic
Exhaustion Controls
Convalescent
Covid
Exhaustion
Immune
Including
Infection
Involves
Long
Pathways
Persistent
Proinflammatory

Semantics

Type Source Name
disease MESH Long COVID
disease MESH severe acute respiratory syndrome coronavirus 2 infection
disease MESH infection
pathway REACTOME Metabolism
disease MESH severe acute respiratory syndrome
disease MESH coronavirus infection
drug DRUGBANK Factor IX Complex (Human)
disease MESH PCC
disease MESH fatigue
disease MESH brain fog
drug DRUGBANK Hydrocortisone
drug DRUGBANK Ritonavir
drug DRUGBANK Coenzyme M
disease MESH varicella
disease MESH zoster
disease MESH Western equine encephalitis
disease MESH herpes simplex
disease MESH influenza
pathway KEGG Nitrogen metabolism
disease MESH death
drug DRUGBANK Amino acids
disease MESH inflammation
disease MESH included
disease MESH pain
disease MESH cough

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